Atlas of Ocular Infections, Ocular Infections, Textbook of Diagnostic Microbiology 4th edition 2011, Connie R. Mahon, Donald C. Lehman and George Manuselis, tuyenlab.net, atlas for medical, atlas for Microbiology
|Fig 1. Common ocular structures.|
|Fig 2. Gonococcal conjunctivitis. Note the copious|
discharge in response to invasion by Neisseria gonorrhoeae.
|Fig 3. A, White spots on the conjunctiva represent pockets of Chlamydia|
organisms in tissue. B, Immunofluorescence stain of scrapings from neonatal
conjunctivitis, confirming the presence of chlamydial elementary bodies.
|Fig 4. Acute hemorrhagic conjunctivitis. The|
etiologic agent is usually enterovirus 70 or coxsackievirus
A24. Other members of the enterovirus group may also be
recovered. Note the heavy conjunctival hemorrhaging.
|Fig 5. A, Corneal melt caused by bacterial invasion. B, C streaks of|
Staphylococcus aureus from infected cornea. The enzymes produced by some strains of
S. aureus and Pseudomonas aeruginosa can liquefy the cornea within 48 hours.
|Fig 6. A, Growth of Pseudomonas aeruginosa from daily-wear (soft) contact lens.|
Patient had an ulcerative keratitis. The corneal culture also grew P. aeruginosa. B, Recent
trends in bacteria recovered from contact lenses and solutions.
|Fig 7. Colonies of Mycobacterium fortuitum|
growing on infected corneal graft tissue.
|Fig 8. Frequency of common keratitis viral isolates|
in southern Florida. HSV-1, Herpes simplex virus type 1;
HSV-2, herpes simplex virus type 2.
|Fig 9. “Tracts” of Acanthamoeba trophozoites. The|
meandering trophozoites are at the end of the tracts. The
large clusters of organisms contain trophozoites and cysts.
|Fig 10. Gram stain revealing the oval cyst of|
Microsporidia spp. Organisms can also be detected with
Giemsa, acid-fast, and calcofluor white stains.
|Fig 11. Dacryocystitis (infection of the lacrimal sac)|
of the left eye (arrow) in a young child.
|Fig 13. Acute cytomegalovirus retinitis with optic|
nerve involvement in a 40-year-old patient who is HIV
positive. Active viral particles are seen in satellite lesions
temporal to the main infection (yellow).
|Fig 14. Kaposi sarcoma (raised dark spots) on the|
conjunctiva of a patient who has AIDS.
|Fig 15. Toxoplasma gondii trophozoites and cysts in|
retinal tissue. This protozoan has a predilection for ocular
tissue. This disease is now more common in HIV patients
than in the general U.S. population.
|Fig 16. Extruded scleral buckle on blood agar plate|
with growth of Candida albicans.
|Fig 17. Fungal (yeast) biofilm on contact lens.|
Culture grew Candida albicans.
|Fig 18. A, Concretions being expressed from canaliculi. B, “Smashed” and|
stained concretions, revealing gram-positive, slender, branching rods (Actinomyces israelii).
|Fig 19. Giemsa stain of conjunctival epithelial cells|
with chlamydial inclusions (arrow). The Giemsa stain also
provides information on the types and numbers of
inflammatory cells and the condition of the epithelial cells.
|Fig 21. Sabouraud plate with mould and yeast.|
Patient had a mixed fungal keratitis. Top, The superficial
layer of the cornea was infected with a mould (Fusarium
oxysporum). Bottom, The deeper layers were infected with a
yeast (Candida albicans).
Fig 22. A, Contact lens and lens solution on 5% sheep blood agar surrounded bygrowth of Pseudomonas aeruginosa. B, C streaks growing pigmented and nonpigmented
Serratia marcescens. C, Corneal scrapings. (Note: Each row of C streaks represents a
separate corneal scraping.)
|Fig 23. Staphylococcus epidermidis recovered from|
vitreous fluids (drops) on a blood agar plate. Samples may
be inoculated onto a chocolate or blood agar plate and
allowed to dry or may be streaked out as for a routine
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